Antti Nurmi has over 15 years of experience in the life science industry, with the last 10 spent in various roles of scientific leadership for global pharmaceutical companies and academic groups. He earned his PhD from the University of Kuopio in Finland, where his work continues with a focus on brain research and the molecular mechanisms behind stroke pathology.
Dr. Nurmi received his MSc in physiology from the University of Joensuu, Finland, where he specialized in cardiac electrophysiology (particularly ion channel physiology and the use of patch-clamping techniques). He has published on a variety of topics ranging from cardiac ion channel physiology to therapeutic approaches against disease pathology in models of stroke and Alzheimer's disease.
- Deficiency of prolyl oligopeptidase in mice disturbs synaptic plasticity and reduces anxiety-like behavior, body weight, and brain volume
- The radical scavenger IAC (bis(1-hydroxy-2,2,6,6-tetramethyl-4-piperidinyl) decantionate) decreases mortality, enhances cognitive functions in water maze and reduces amyloid plaque burden in hAβPP transgenic mice